Interaction of EGFR to δ-catenin leads to δ-catenin phosphorylation and enhances EGFR signaling

نویسندگان

  • Yongfeng He
  • Taeyong Ryu
  • Nensi Shrestha
  • Tingting Yuan
  • Hangun Kim
  • Hridaya Shrestha
  • Young-Chang Cho
  • Young-Woo Seo
  • Woo Keun Song
  • Kwonseop Kim
چکیده

Expression of δ-catenin reportedly increases during late stage prostate cancer. Furthermore, it has been demonstrated that expression of EGFR is enhanced in hormone refractory prostate cancer. In this study, we investigated the possible correlation between EGFR and δ-catenin in prostate cancer cells. We found that EGFR interacted with δ-catenin and the interaction decreased in the presence of EGF. We also demonstrated that, on one hand, EGFR phosphorylated δ-catenin in a Src independent manner in the presence of EGF and on the other hand, δ-catenin enhanced protein stability of EGFR and strengthened the EGFR/Erk1/2 signaling pathway. Our findings added a new perspective to the interaction of EGFR to the E-cadherin complex. They also provided novel insights to the roles of δ-catenin in prostate cancer cells.

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2016